Weighing Evidence from Mendelian Randomization—Early-Life Obesity as a Causal Factor in Multiple Sclerosis?
Copyright policy )Weighing Evidence from Mendelian Randomization—Early-Life Obesity as a Causal Factor in Multiple Sclerosis?
In this Perspective, Alberto Ascherio and Kassandra Munger discuss the implications of Richards and colleagues' study exploring the role of early-life obesity in risk of multiple sclerosis.
- Harvard University United States
- Harvard T.H. Chan School of Public Health United States
- HARVARD SCHOOL OF PUBLIC HEALTH
Multiple Sclerosis, Physiology, Epidemiology, Immunology, 610, Polymorphism, Single Nucleotide, Autoimmune Diseases, Body Mass Index, Dose Prediction Methods, 616, Medicine and Health Sciences, Genome-Wide Association Studies, Genetics, Humans, Prospective Studies, Obesity, Pharmaceutics, Body Weight, R, Biology and Life Sciences, Computational Biology, Neurodegenerative Diseases, Human Genetics, Genomics, Mendelian Randomization Analysis, Genome Analysis, Demyelinating Disorders, Physiological Parameters, Neurology, Genetic Epidemiology, Perspective, Observational Studies, Medicine, Clinical Immunology, Clinical Medicine, Genome-Wide Association Study
Multiple Sclerosis, Physiology, Epidemiology, Immunology, 610, Polymorphism, Single Nucleotide, Autoimmune Diseases, Body Mass Index, Dose Prediction Methods, 616, Medicine and Health Sciences, Genome-Wide Association Studies, Genetics, Humans, Prospective Studies, Obesity, Pharmaceutics, Body Weight, R, Biology and Life Sciences, Computational Biology, Neurodegenerative Diseases, Human Genetics, Genomics, Mendelian Randomization Analysis, Genome Analysis, Demyelinating Disorders, Physiological Parameters, Neurology, Genetic Epidemiology, Perspective, Observational Studies, Medicine, Clinical Immunology, Clinical Medicine, Genome-Wide Association Study
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