Brx Mediates the Response of Lymphocytes to Osmotic Stress Through the Activation of NFAT5
Brx Mediates the Response of Lymphocytes to Osmotic Stress Through the Activation of NFAT5
The guanine nucleotide exchange factor Brx is essential for osmotic stress–mediated expression of nfat5 in lymphocytes.
- National Institute of Health Pakistan
- Washington DC VA Medical Center United States
- National Cancer Institute United States
- George Washington University United States
- Veterans Health Administration United States
Mice, Knockout, B-Lymphocytes, Dehydration, A Kinase Anchor Proteins, Immunoglobulins, Protein-Tyrosine Kinases, Minor Histocompatibility Antigens, Mice, Gene Expression Regulation, Osmotic Pressure, Proto-Oncogene Proteins, B-Cell Activating Factor, Animals, Humans, Lymphocytes, RNA, Messenger, Spleen, Transcription Factors
Mice, Knockout, B-Lymphocytes, Dehydration, A Kinase Anchor Proteins, Immunoglobulins, Protein-Tyrosine Kinases, Minor Histocompatibility Antigens, Mice, Gene Expression Regulation, Osmotic Pressure, Proto-Oncogene Proteins, B-Cell Activating Factor, Animals, Humans, Lymphocytes, RNA, Messenger, Spleen, Transcription Factors
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