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A mouse model of 22q11.2 deletions: Molecular and behavioral signatures of Parkinson’s disease and schizophrenia

descriptionPublicationkeyboard_double_arrow_right Article , Other literature type 03 Aug 2018 Japan English Publisher:American Association for the Advancement of Science (AAAS)Journal:Science Advances, volume 4 (eissn: 2375-2548,

Authors: Nancy J. Butcher; Anne S. Bassett; Anne S. Bassett; Frederick C. Nucifora; Akiko Sumitomo; Takeshi Sakurai; Akira Sawa; +5 Authors

doi: 10.1126/sciadv.aar6637

pmid: 30116778

pmc: PMC6093626

handle: 2433/234197

A mouse model of 22q11.2 deletions: Molecular and behavioral signatures of Parkinson’s disease and schizophrenia

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Abstract

22q11.2 deletions, a genetic risk for schizophrenia, could be susceptible to Parkinson’s disease through elevated expression of α-synuclein.

Country

Japan

Related Organizations

Kyoto University
Japan
University of Toronto
Canada
Johns Hopkins Medicine
United States
Johns Hopkins University School of Medicine
United States
University Health Network
Canada

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Keywords

Male, Mice, Knockout, Behavior, Animal, Chromosomes, Human, Pair 22, Parkinson Disease, Mice, Inbred C57BL, Disease Models, Animal, Mice, DiGeorge Syndrome, Schizophrenia, alpha-Synuclein, Animals, Humans, Research Articles, Sequence Deletion

2 Research products, page 1 of 1

Altered expression of hippocampal dentate granule neuron genes in a mouse model of human 22q11 deletion syndrome
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A mouse model of 22q11.2 deletions: Molecular and behavioral signatures of Parkinson’s disease and schizophrenia

A mouse model of 22q11.2 deletions: Molecular and behavioral signatures of Parkinson’s disease and schizophrenia

2 Research products, page 1 of 1

Altered expression of hippocampal dentate granule neuron genes in a mouse model of human 22q11 deletion syndrome

A mouse model of 22q11.2 deletions: Molecular and behavioral signatures of Parkinson's disease and schizophrenia