descriptionPublicationkeyboard_double_arrow_right Article 25 Aug 2011 English Publisher:American Society of HematologyJournal:Blood, volume 118, pages 2,029-2,030 (issn: 0006-4971, eissn: 1528-0020,

Authors: BALDUINI, ALESSANDRA;

doi: 10.1182/blood-2011-06-361303

pmid: 21868582

handle: 11571/280514

New aspects of Filamin A versatility

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Abstract

Filamins are crucial for human development and they may play a role in the regulation of megakaryocyte maturation. Still, little is known about the molecular mechanisms involved in platelet production from megakaryocytes and, consequently, how those mechanisms impact the pathogenesis of platelet

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University of Pavia
Italy
Tufts University
United States

8 Research products, page 1 of 1

Impaired Viability, Platelet Survival, Morphology and Function in Mice Lacking Filamin A.
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Filamin A Deficiency in Platelets Reveals Functional Impairment in ITAM-Based Signaling.
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FlnA binding to PACSIN2 F-BAR domain regulates membrane tubulation in megakaryocytes and platelets
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Deletion of Filamin A in Monocytes Protects Cortical and Trabecular Bone from Post-menopausal Changes in Bone Microarchitecture
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Filamin A regulates monocyte migration through Rho small GTPases during osteoclastogenesis
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A novel interaction between FlnA and Syk regulates platelet ITAM-mediated receptor signaling and function
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FlnA-null megakaryocytes prematurely release large and fragile platelets that circulate poorly
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PACSIN2: a BAR-rier forming the megakaryocyte DMS
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