HVEM signaling promotes protective antibody-dependent cellular cytotoxicity (ADCC) vaccine responses to herpes simplex viruses
NIH| HVEM-BTLA system in Inflammation ,
NIH| Innate Immune Responses and Vaccines Against Tumor-Associated Herpesviruses ,
NIH| Host-virus interactions in the control of filovirus entry
Authors: Clare Burn Aschner; Lip Nam Loh; Benjamin Galen; Isabel Delwel; Rohit K. Jangra; Scott J. Garforth; Kartik Chandran; +4 Authors
Clare Burn Aschner; Lip Nam Loh; Benjamin Galen; Isabel Delwel; Rohit K. Jangra; Scott J. Garforth; Kartik Chandran; Steven Almo; William R. Jacobs; Carl F. Ware; Betsy C. Herold;
HVEM signaling promotes protective antibody-dependent cellular cytotoxicity (ADCC) vaccine responses to herpes simplex viruses
Abstract
Herpes simplex virus glycoprotein D allows viral evasion of protective host ADCC responses by interfering with LIGHT-HVEM signaling.
Related Organizations
Keywords
Male, Mice, Knockout, Antibody-Dependent Cell Cytotoxicity, Herpes Simplex, Viral Vaccines, Mice, Inbred C57BL, Animals, Simplexvirus, Female, Receptors, Tumor Necrosis Factor, Member 14, Signal Transduction
Male, Mice, Knockout, Antibody-Dependent Cell Cytotoxicity, Herpes Simplex, Viral Vaccines, Mice, Inbred C57BL, Animals, Simplexvirus, Female, Receptors, Tumor Necrosis Factor, Member 14, Signal Transduction
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citations
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This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
popularity
Popularity provided by BIP!
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
16
Top 10%
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Top 10%
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