TSLP Elicits IL-33–Independent Innate Lymphoid Cell Responses to Promote Skin Inflammation
Copyright policy )SNSF| Cellular and Molecular Mechanisms in Food Allergy
Kim Brian S.; Siracusa Mark C.; Saenz Steven A.; Noti Mario; Monticelli Laurel A.; Sonnenberg Gregory F.; Hepworth Matthew R.; +3 Authors
Kim Brian S.; Siracusa Mark C.; Saenz Steven A.; Noti Mario; Monticelli Laurel A.; Sonnenberg Gregory F.; Hepworth Matthew R.; Van Voorhees Abby S.; Comeau Michael R.; Artis David;
TSLP Elicits IL-33–Independent Innate Lymphoid Cell Responses to Promote Skin Inflammation
Group 2 innate lymphoid cells are essential to the pathogenesis of atopic dermatitis–like disease in a TSLP-dependent, IL-33–independent manner.
- Amgen (United States) United States
- University of Salford United Kingdom
- University of Pennsylvania United States
Inflammation, Interleukins, Adaptive Immunity, Interleukin-33, Immunity, Innate, Dermatitis, Atopic, Mice, Inbred C57BL, Disease Models, Animal, Mice, Thymic Stromal Lymphopoietin, Animals, Cytokines, Humans, Lymphocytes, Protein Binding, Skin
Inflammation, Interleukins, Adaptive Immunity, Interleukin-33, Immunity, Innate, Dermatitis, Atopic, Mice, Inbred C57BL, Disease Models, Animal, Mice, Thymic Stromal Lymphopoietin, Animals, Cytokines, Humans, Lymphocytes, Protein Binding, Skin
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This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
Citations provided by BIP!popularityPopularity provided by BIP!
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
Popularity provided by BIP! 656
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