Localisation of a dystrophin-related autosomal gene to 6q24 in man, and to mouse chromosome 10 in the region of the dystrophia muscularis (dy) locus
Copyright policy )Localisation of a dystrophin-related autosomal gene to 6q24 in man, and to mouse chromosome 10 in the region of the dystrophia muscularis (dy) locus
We have localised a dystrophin-related autosomal gene called DMDL (Duchenne muscular dystrophy-like) to human chromosome 61q24 by in situ hybridisation. Using restriction fragment length polymorphism analysis in two mouse species, we have localised the homologous gene Dmdl in the mouse to chromosome 10 proximal to the Myb oncogene. A neuromuscular disease locus dystrophia muscularis (dy) has previously been assigned to this region of mouse chromosome 10.
- Institut Pasteur France
- Northamptonshire Healthcare NHS Foundation Trust United Kingdom
- Oxford University Hospitals NHS Trust United Kingdom
- John Radcliffe Hospital United Kingdom
Male, Mice, Inbred BALB C, Genetic Linkage, Chromosome Mapping, Muscle Proteins, Mice, Inbred Strains, Muscular Dystrophy, Animal, Muscular Dystrophies, Dystrophin, Mice, Inbred C57BL, Mice, Animals, Humans, Chromosomes, Human, Pair 6
Male, Mice, Inbred BALB C, Genetic Linkage, Chromosome Mapping, Muscle Proteins, Mice, Inbred Strains, Muscular Dystrophy, Animal, Muscular Dystrophies, Dystrophin, Mice, Inbred C57BL, Mice, Animals, Humans, Chromosomes, Human, Pair 6
citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).80 popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.Average influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).Top 10% impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.Top 1%
Citations provided by BIP!Popularity provided by BIP!