Reversal of Disease-Related Pathologies in the Fragile X Mouse Model by Selective Activation of GABABReceptors with Arbaclofen
Copyright policy )Reversal of Disease-Related Pathologies in the Fragile X Mouse Model by Selective Activation of GABABReceptors with Arbaclofen
Pharmacological activation of the GABABreceptor with arbaclofen in a mouse model of fragile X syndrome corrects neuronal defects associated with the disease.
- Baylor College of Medicine United States
- Massachusetts Institute of Technology United States
- University of Edinburgh United Kingdom
- Scripps Research Institute United States
- Emory University United States
Male, Mice, Knockout, Baclofen, Behavior, Animal, Dendritic Spines, Drinking Water, Hippocampus, Disease Models, Animal, Fragile X Mental Retardation Protein, Mice, Protein Transport, Phenotype, Receptors, GABA-B, Seizures, Fragile X Syndrome, GABA-B Receptor Agonists, Polyribosomes, Protein Biosynthesis, Animals, Receptors, AMPA
Male, Mice, Knockout, Baclofen, Behavior, Animal, Dendritic Spines, Drinking Water, Hippocampus, Disease Models, Animal, Fragile X Mental Retardation Protein, Mice, Protein Transport, Phenotype, Receptors, GABA-B, Seizures, Fragile X Syndrome, GABA-B Receptor Agonists, Polyribosomes, Protein Biosynthesis, Animals, Receptors, AMPA
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