Additional file 5: of Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical WNT signaling
NIH| Mechanism of PIN development in Abi1/Hssh3bp1 KO mouse
Authors: Nath, Disharee; Li, Xiang; Mondragon, Claudia; Post, Dawn; Chen, Ming; White, Julie; Hryniewicz-Jankowska, Anita; +12 Authors
Nath, Disharee; Li, Xiang; Mondragon, Claudia; Post, Dawn; Chen, Ming; White, Julie; Hryniewicz-Jankowska, Anita; Caza, Tiffany; Kuznetsov, Vladimir; Hehnly, Heidi; Jamaspishvili, Tamara; Berman, David; Zhang, Fan; Kung, Sonia; Fazli, Ladan; Gleave, Martin; Bratslavsky, Gennady; Pandolfi, Pier; Kotula, Leszek;
Additional file 5: of Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical WNT signaling
Abstract
Figure S5. Generated ABI1 KO cells exhibit no change in p-β-catenin or N-WASP. (A) Representative western blots showing no change in p-β-catenin, total β-catenin or N-WASP upon ABI1 loss. Blot shows parental RWPE-1 cells, two control clones and three ABI1 KO clones. β-Actin was used as the loading control. (JPG 461 kb)
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