MicroRNAs Differentially Regulated by Akt Isoforms Control EMT and Stem Cell Renewal in Cancer Cells
Copyright policy )NIH| Genomic targets of oncoproteins and tumor suppressors ,
NIH| Akt in T cell development and function
Dimitrios, Iliopoulos; Christos, Polytarchou; Maria, Hatziapostolou; Filippos, Kottakis; Ioanna G, Maroulakou; Kevin, Struhl; Philip N, Tsichlis;
MicroRNAs Differentially Regulated by Akt Isoforms Control EMT and Stem Cell Renewal in Cancer Cells
Akt-dependent induction of a metastatic phenotype may depend on the balance of Akt1 and Akt2.
- TUFTS MEDICAL CENTER
- Department of Biological Chemistry and Molecular Pharmacology Harvard Medical School United States
- Harvard Medical School United States
- Tufts Medical Center United States
- Molecular Research Institute United States
Stem Cells, Carcinoma, Cadherins, Epithelium, Gene Expression Regulation, Neoplastic, Mesoderm, Mice, MicroRNAs, Transforming Growth Factor beta, Cell Line, Tumor, Animals, Humans, Protein Isoforms, Neoplasm Metastasis, Proto-Oncogene Proteins c-akt
Stem Cells, Carcinoma, Cadherins, Epithelium, Gene Expression Regulation, Neoplastic, Mesoderm, Mice, MicroRNAs, Transforming Growth Factor beta, Cell Line, Tumor, Animals, Humans, Protein Isoforms, Neoplasm Metastasis, Proto-Oncogene Proteins c-akt
citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).200 popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.Top 10% influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).Top 10% impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.Top 1%
citations
Citations provided by BIP!
This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).
Citations provided by BIP!popularityPopularity provided by BIP!
This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.
Popularity provided by BIP! 200
Top 10%
Top 10%
Top 1%
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