Mcl‐1128–350 fragment induces apoptosis through direct interaction with Bax
Copyright policy )Mcl‐1128–350 fragment induces apoptosis through direct interaction with Bax
MINT‐7307171: F1 ATPase (uniprotkb:Q5TC12), Mcl‐1 (uniprotkb:Q07820) and BAX (uniprotkb:Q07812) colocalize (MI:0403) by cosedimentation through density gradients (MI:0029)
- University of Nantes France
- Centre Hospitalier Universitaire de Nantes France
- Ligue Nationale Contre le Cancer France
- University of Nantes France
Cleaved Mcl-1, Blotting, Western, Mcl-1, Apoptosis, Bax activation, Cell Line, Mitochondria, bcl-2 Homologous Antagonist-Killer Protein, Proto-Oncogene Proteins c-bcl-2, Cell Line, Tumor, Humans, Immunoprecipitation, Myeloid Cell Leukemia Sequence 1 Protein, Plasmids, Protein Binding, bcl-2-Associated X Protein
Cleaved Mcl-1, Blotting, Western, Mcl-1, Apoptosis, Bax activation, Cell Line, Mitochondria, bcl-2 Homologous Antagonist-Killer Protein, Proto-Oncogene Proteins c-bcl-2, Cell Line, Tumor, Humans, Immunoprecipitation, Myeloid Cell Leukemia Sequence 1 Protein, Plasmids, Protein Binding, bcl-2-Associated X Protein
citations This is an alternative to the "Influence" indicator, which also reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).12 popularity This indicator reflects the "current" impact/attention (the "hype") of an article in the research community at large, based on the underlying citation network.Average influence This indicator reflects the overall/total impact of an article in the research community at large, based on the underlying citation network (diachronically).Average impulse This indicator reflects the initial momentum of an article directly after its publication, based on the underlying citation network.Top 10%
Citations provided by BIP!Popularity provided by BIP!